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Before I was switched to Amantadine I was unable to walk, dress myself and perform routine operations. My neurologist first prescribed me with another medication but I developed severe migraine attacks with that stuff, don't even remember if there was any positive effect on my Parkinson. Then I was put on Amantadine and this is a god sent! So happy that my doctor chose this medication for me! With this drug it is naturally easier to live with the diagnosis and the symptoms are fading away. I can't say Amantadine cures the disease but it makes it tolerable. I can cope without anybody's help most of the time. It is important for a man of 50 years old.
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For me Amantadine is like oxygen - essential for life! This medication is the only solution on the market that helps me cope with my Parkinson disease. Provided taken regularly and in accordance with the prescribed regimen, Amantadine produces excellent results. Before this drug I have been on several other medications and they were more or less effective, with certain positive effects. But they make no comparison for Amantadine. With this medication I can control myself almost 100%. No more tremors, no memory lapses, no panic attacks. The difference is so obvious, I would pray for the people who have invented this medication! My doctor is also pleased with the results and he says my case is the most successful in his practice.

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Amantadine has multiple mechanisms of action, including release of dopamine and norepinephrine from nerve endings, acting as a weak N-methyl- d -aspartate receptor antagonist, and anticholinergic effects.

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The proportion each drug-cell interaction was significant among all gene set sizes provided a relative weight for each predicted interaction ().

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The patient should be observed for hyperactivity and convulsions; if required, sedation, and anticonvulsant therapy should be administered.

Extended release compounds useful in minimizing side effects.

The drug was well tolerated.

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Catatonia: clinical aspects and neurobiological correlates.

Most people with PD did not inherit the disease in their genes.

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Catecholamine agonists appear to be more useful as tools to elucidate the pathophysiology of TD than as reliable treatments for patients.

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Following oral administration of 200 mg of 14C-bupropion in humans, 87% and 10% of the radioactive dose were recovered in the urine and feces, respectively.

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It concluded it might be useful for prevention in high risk persons who had not been vaccinated.

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Subsequent to body mass reduction, AHI should be reassessed using in-lab polysomnography to determine whether PAP adjustments are needed or if PAP therapy may be discontinued altogether.

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Because of scientific advances, improvements in genetic testing, and increased awareness, the number of children diagnosed with Fragile X has increased significantly over the last decade.

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Use of oseltamivir in 12 European countries between 2002 and 2007---lack of association with the appearance of oseltamivir-resistant influenza A(H1N1) viruses.

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Participants were recruited to the study at the time of their simulation visit and completed baseline questionnaires.

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Choice C is incorrect because an adverse effect of amantadine is a mottled discoloration of the skin.

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